Hemophilia A
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Coagulation
- Common Pathway of Coagulation
- Extrinsic Pathway of Coagulation
- Intrinsic Pathway of Coagulation
- Hemophilia A
- Hemophilia B
- Hemophilia C
- Factor V Leiden
- Antithrombin-3 Deficiency
Summary
Hemophilia A is a X-linked recessive bleeding disorder caused by a deficiency in factor 8. Patients with hemophilia A present with easy bruising and bleeding, including bleeding into the joint spaces and muscles. Lab testing in patients with hemophilia A will reveal decreased levels of factor 8 and an increased PTT. Treatment includes desmopressin for milder cases, although severe cases will require factor 8 replacement.
Key Points
- Hemophilia A
- Pathogenesis
- Deficiency of Factor VIII
- Leads to inadequate generation of thrombin (Factor IIa) and improper coagulation of blood
- Inhibits intrinsic coagulation cascade
- Deficiency of Factor VIII
- Genetics
- XLR inheritance
- Almost exclusively affects males
- XLR inheritance
- Presentation
- Clinical symptoms indistinguishable from other hemophilias
- Bleeding
- Hemarthrosis - bleeding into joints
- Intramuscular hemorrhage
- Easy bruising
- Bleeding after trauma or surgery (e.g. dental procedure)
- Diagnosis
- Definitive: Factor VIII levels
- Elevated PTT
- Due to inhibited intrinsic coagulation pathway
- Normal PT
- Extrinsic (tissue factor) pathway is unaffected
- Normal Bleeding Time
- Platelet plug formation (primary hemostasis is unaffected)
- Treatment
- ddAVP (desmopressin)
- Increases endogenous factor VIII, since vWF is a carrier that stabilizes circulating Factor VIII
- Used for mild cases
- Recombinant Factor VIII replacement
- Used for severe cases
- ddAVP (desmopressin)
- Pathogenesis