TNF-alpha
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Cytokines
Summary
Tumor necrosis factor-alpha (TNFα), also known as cachexin, or cachectin, is one of the chief cytokines involved in systemic inflammation and the acute phase reaction. As a cell signalling protein, it activates many other types of immune cells to potentiate inflammation. As a pyrogen, it works on hypothalamic nuclei to increase body temperature. TNF-alpha secreted by tumors is also implicated in paraneoplastic cachexia syndrome.
Key Points
- Tumor Necrosis Factor-alpha (TNF-alpha)
- Secreted by Macrophages
- As well as mast cells, to a lesser degree
- Actions
- Induces fever
- Signalling at hypothalamic nuclei increase body temperature
- Promotes inflammation
- Along with IL-1 and IL-6, TNF-alpha induces the systemic inflammatory response (SIRS) and sepsis, and causes hepatic release of acute-phase reactants (e.g. C-reactive protein and fibrinogen).
- Activates endothelium to increase vascular leak and causes WBC recruitment
- Implicated in paraneoplastic cachexia
- TNF-alpha secreted by tumors
- Target of many anti-inflammatory or anti-rheumatic drugs
- Infliximab (chimeric monoclonal antibody)
- Certolizumab (pegylated humanized antibody)
- Etanercept (recombinant decoy receptor)
- Induces fever
- Secreted by Macrophages